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Curriculum Vitae, Sara Ann Mills, M.D., Ph.D.

Education

1972-1976: B.A. Biology, Grinnell College, Grinnell, Iowa

1977-1979: B.S.N., Nursing, University of Arizona, Tucson, Arizona

1981-1987: Ph.D., Medical Sciences (Pharmacology) University of New Mexico,
Albuquerque, New Mexico

1987-1990: M.D., University of New Mexico, Albuquerque, New Mexico

1990-1994: Residency (Dermatology), Emory University, Atlanta, Georgia

Board Certification

1995: American Board of Dermatology

2004: Recertification: American Board of Dermatology

2013: Recertification: American Board of Dermatology

Honors and Awards

1975-1976: Mortar Board, Grinnell College

1978-1979: Phi Beta Kappa (Scholastic Honorary), University of Arizona

1978-1979: Sigma Theta Tau (Nursing Honorary), University of Arizona

1977-1979: Dean's List, University of Arizona

1979: President's Scholarship Award, University of Arizona

1979: Graduation with High Distinction, University of Arizona

1985-1986: Medical Sciences Graduate Student Tuition Fellowship

1986-1987: Legislative Neuroscience Stipend Award Grant

1986: Medical Student Research Award, 2nd Place, Poster Division University of
New Mexico

1989: Alpha Omega Alpha (Medical Student Honor Society)

Professional Societies

1984-1990: Society of Neuroscience

1987-1990: American Medical Association

1991-present: American Academy of Dermatology

1991-1994: Atlanta Dermatology Association

1994-present: New Mexico Dermatologic Society, President 2002

1994-1996: Women's Dermatologic Society

1996-present: Contact Dermatitis Society

1997-present: American Society for Mohs Surgery

Employment

1976-1977: Nursing Assistant, Tucson Medical Center, Tucson, Arizona

1976-1980: R.N., University of Arizona Health Sciences Center, Tucson, Arizona
(SICU)

1982-1986: Graduate Research Assistant, University of New Mexico

1983-1986: R.N., Kaseman Presbyterian Hospital, Albuquerque, New Mexico
(Subacute Care, ICU)

1990-1991: Internship-Internal Medicine, University of New Mexico, Albuquerque,
New Mexico

1991-1994: Residency-Dermatology, Emory University, Atlanta, Georgia; Chief
Resident 1993-1994

1994-1998: Dermatology Consultants of Albuquerque, Albuquerque, New Mexico

1998-present: Western Dermatology Consultants and the Spa at WDC, Albuquerque,
New Mexico

Publications

  • Mills, Sara A. and Savage, D. D.: Evidence of neonatal hypothyroidism in genetically epilepsy-prone rats. Epilepsy Research 2: 102-110, 1988.
  • Savage, Daniel D.; Mills, S.A; Jobe, P.C.; and Reigel, C.E.: Elevation of 3H-dihydromorphine binding sites in hipposcampal formation of genetically epilepsy-prone rat. Life Sci. 43: 239-246, 1988.
  • Mills, Sara A; Razani-Boroujerdi, S; Reigel, C.E.; Jobe, P.C.; and Savage, D.D: Decrease in hipposcampal 3H-vinylidene kainic acid binding sites in genetically epilepsy-prone rats. Neuroscience. 35(3): 519-524, 1990.

Abstracts

  • Mills, Sara A.; Reigel, C.E.; Jobe, P.C.; and Savage, D.D.: Increase in the number of hipposcampal 3H-glutamate binding sites in rats inbred for genetic predisposition to acoustic stimulus induced seizures. Soc. Neurosci. Abstracts 11:1315, 1985.
  • Mills, Sara A. and Savage, D. D.: Evidence of neonatal hypothyroidism in genetically epilepsy-prone rats. Soc. Neurosci. Abstracts 12:72, 1986.
  • Savage, Daniel D.; Mills, S. A.; Reigel, C. E.; and Jobe, P.C.: Radiohistochemical study of alterations in specific 3H-glutamate and 3H-vinylidene kainic acid binding sites in hippocampal formation of genetically epilepsy-prone rat. Soc. Neurosci. Abstracts 12:72, 1986
  • Savage, Daniel D.; Mills, S.A.; Jobe, P.C.; and Reigel, C. E.: Elevation of 3H-dihydromorphine binding sites in hippocampal formation of genetically epilepsy-prone rat. Fed. Proc. 46:706, 1987.
  • Mills, Sara A.; Reigel, C. E.; Jobe, P.C.; and Savage, D. D.: Deficits in serum growth hormone and postnatal growth in the genetically epilepsy-prone rat. Soc. Neurosci. Abstracts 13:943, 1987.

PH.D. Dissertation: 1987

The Genetically Epilepsy-Prone Rat: Evidence implicating endocrine deficits and limbic neurotransmitter systems in the development of the seizure-prone state.